Search for: [Abstract = "vity was higher in ASgroup than in controls \(all p<0.001\). In AS patients TAFI activity remained unaltered at peak exercise, but then after a decrease after one hour it rose above the baseline value \(p=0.003\). In controls TAFI activity was reduced during the test and after its completion compared to the baseline \(all p<0.01,\). Of note there was a weak association between TAFI antigen and Vmax \(r=0.36, p=0.042\). Importantly, in AS patients CLT at baseline, but not in three other time points, correlated with TAFI activity \(r=0.69, p<0.001\), α2\-AP \(r=0.47, p=0.007\) and plasminogen \(r=\-0.55, p=0.001\). In controls CLT correlated with TAFI activity in all time points \(r=0.65, p<0.001\; r= 0.54, p=0.001\; r=0.42, p=0.015\; r=0.50, p=0.003, respectively\).Plasminogen was similar at baseline in both groups, but then plasminogen became higher in AS group \(all p<0.05, respectively\). There were no differences in PAI\-1 antigen between the two groups in any time point. Although α2\- antiplasmin remained unaltered during exercise test in the AS group, it was higher than in the control group at baseline \(p=0.024\), similar at peak exercise, and lower at one and 24 hours post exercise \(both p<0.05\). Importantly, in AS patients CLT at baseline, but not in three other time points, correlated with TAFI activity \(r=0.69, p<0.001\), α2\- antiplasmin \(r=0.47, p=0.007\) and plasminogen \(r=\-0.55, p=0.001\). In c"]

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