Search for: [Abstract = "ed using SAS, version 9.1. Results\: All three genotypes were in compliance with Hardy\-Weinberg equilibrium. The frequency of T allele at position 276 among non\-diabetic \(20%\) versus DM2 \(15%\) subjects was significantly higher. The T allele was significantly associated\(p<0,05\) with lower insulin resistance \(HOMA\-IR, fasting insulin\) among non\-diabetic subjects. T allele carriers among DM2 subjects rarely suffered from hypertension \(p<0,005\). An allele at position \-11391 was significantly associated \(p<0,05\) with higher insulin resistance \(HOMA\-IR, fasting insulin\) and an higher LDL level. L\/A ratio and fasting insulin were higher among carriers of the G allele at position 45 \(p<0,05\). The OLTT revealed higher postprandial insulin levels in G allele carriers at position 45 \(p<0,05\). Adjustments for gender, BMI and age were included in every analysis. Comparing the results with haplotype analysis the effect of SNPs was confirmed. Conclusions\: This study demonstrates the link between metabolic syndrome phenotypes and adiponectin polymorphisms\: 45T>G, 276G>T, \-11391G>A in the population of the South of Poland. Allele G at position 276, allele A at position \-11391 and allele T at position 45 seem to be markers of the risk of metabolic syndrome."]

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