Search for: [Abstract = "The theories formulated to explain atherogenesis have been evolving from vessel wall lipid accumulation to endothelial dysfunction with vascular wall remodelling. The progress in basic and experimental science have shown that inflammatory and immunological processes, cell and molecular machanizm can initiate atherogenesis. High frequency of cardiac and vessel disease creating on the ground of atherosclerosis caused that prevention is one of major aim of medicine. Estimating the risk of coronary artery disease and coming of clinicals incidents is the basic aim of intervention work. Markers of endothelian demage and activation inflammatory response have a prognostic value independent of clasic risk factors. Diagnostic these markers, although still limited, shought become the basic way of identification patients, that are the most danger with aterosclerosis complications. In the study tried to estimate the level of anti\-hsp60 antibody, C\-reactive protein, fibrinogen, leucocytes and anti\-Ch.p. in class IgG and IgA in group with stable coronary artery disease \(CAD\) and in group with healthy subjects. In the research comperized the level of anti\-hsp60 antibodies, C\-reactive protein, fibrinogen, leucocytes and anti\-Ch.p. in class IgG and IgA in depending on amount of vessels involved in atherosclerosis and progressing atherosclerosis according to classyfication of Canadian Cardiologic"]

Number of results: 1