Search for: [Abstract = "The aim\: Neurogenic inflammation may contribute to the \(max 1400 znak6w\) pathogenesis of migraine, but not episodic tension\-type headache \(ETTH\). The aim of this research was to establish whether in children during migraine and ETTH the level of IL\-1 beta, IL\-6, TNF and soluble TNF receptor type I \(sTNFRI\) was changed, and whether the level of those cytokines in headache\-free period differs between groups with headache and controls. Material\: 89 children aged 6\-17 years were included \( 16 with migraine with aura, 14 without aura, 31 with ETTH and 28 controls without headaches\). Methods\: In children with headaches, the blood was sampled in the first hour and in 3 and in 6 hour of the headache, 6 hours after pain termination, in the headachefree period, and in controls under the same conditions. Results\: No differences in cytokine levels were detected during headache attack between groups with migraine and ETTH and during headache\-free interval between groups of headache and controls. In 12\/30 children with migraine the significant increase in IL\-6 level was detected in the first hour of attack. The level of IL\-1 beta was higher in migraine without aura during headache\-free period and in the 1. hour of attack, TNF level was higher in this group 6 hours after pain termination, as compared with migraine with aura. There was significant increase in IL\-6 level"]

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