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Search for: [Abstract = "Summary\: Morbidity and mortality from cardiovascular diseases increase rapidly in women after menopause. Past studies indicate a decrease of 17β\-oestradiol secretion as an important factor in the pathogenesis of ischaemic heart disease and arterial hypertension. There are some evidences that after menopause the autonomic nervous system dysfunction is the cause of inappropriate regulation of cardiovascular system and its augmented susceptibility for environmental factors. Neuroendocrinic disturbances are strictly connected with metabolic disturbances, and commonly are involved in the development of hypertension. Probably adipocytokines are the important factors binding the sympathetic overactivity with occurrence of hypertension. Some evidences from clinical and experimental studies have inclined to evaluate the participation of these parameters in the pathogenesis of hypertension. Similar role is attributed to brain natriuretic peptide \(BNP\), although actually only limited studies about its role in patients with mild hypertension exist. There are no evidences about the influence of menopause on BNP level. Present studies indicate to the important prognostic value of these measurements \- BNP, adiponectin, leptin and C\-reactive protein \(CRP\) together in evaluation of cardiovascular risk. The aim of this study was evaluation of autonomic nervous system activity in women after men"]

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