Search for: [Abstract = "On average, there was 1.73 lesion treated per patient. PCI was considered successful in 127 \(96.2%\) patients. During 30 days follow\-up MACE occurred in 3 \(2.4%\) patients, including 1 \(0.8%\) sudden death on 4\-th day after PCI and 2 \(1.6%\) MIs, Following successful PCI, symptom reduction by at least one CCS class was seen in 101 \(80.2%\) pts, including all pts treated in the course of acute coronary syndrome \(ACS\). In the long\-term follow up of 2.5 years \(range 1\-63 months\), MACE occurred in 54 out of 127 pts subjected to PCI. There were 9 deaths, including 5 cardiac deaths. Due to symptom recurrence, another angiography was performed in 44 pts. This revealed restenosis in 26 pts, atherosclerosis progression in 10 pts, and both causes in 2 pts. In 27 pts rePCI was performed, 3 pts were referred for reCABG and 2 for cardiac transplantation. After successful PCI, MACE\-free survival was noted in 81% pts at 1 year and 65% at 5 years. The likelihood of MACE\-free survival was higher for those after nv\-PCI as compared to svg\-PCI \(p=0.047\). There was no difference in MACE\-free survival for those with PCI in stable angina vs. ACS \(89.2 vs. 93.6% at 6 months, 71.9 vs. 62.5% at 2 years\). Among the 127 pts subjected to successful PCI, 64 \(50.4%\) had hs\-CRP ≥ 3.5mg\/dl and 47 \(37%\) fibrinogen >3.5 g\/L. No relationship was revealed between non\-specific inflammatory markers elevation \(including hs\-CRP\) and the risk of MACE. Patients with elevated IgA or IgG against ChP had a higher risk of MACE \(particularly death\) \(p=0.047 and p=0.045 respectively\). To determine independent predictors of long\-term MACE, 44 parameters were included in the Cox analysis. This showed the following independent factors\: left main coronary artery PCI \(OR 3.1\), svg\-PCI \(OR 2.1\) and anti\-ChP IgA >12 EIU \(OR 1.9\).During a mean follow\-up of 7.5 years after CABG, bypass graft degeneration was found in 64% pts and it was the main cause of angina recurrence. Native vessel atherosclerosis progression occurred in 44% pts\; this was manifest clinically significantly earlier than bypass graft degeneration. Atherosclerosis progression in native vessels, lack of IMA use as a graft, hyperlidaemia, and elevated white blood count were identified as independent factors of earlier angina recurrence after CABG. The PCI procedure was feasible in \~3\/4 of pts with angina recurrence after CABG. PCI was safe and successful in symptom resolution. Following PCI for nv atherosclerosis progression or bypass graft degeneration, MACE occurred in \~1\/3 pts. During a mean follow\-up of 2.5 years, one in four pts required another intervention. In this period, \~50% pts required hospitalization for various reasons \(including e.g., angina, atrial fibrillation of heart failure\). The probability of MACE was higher following bypass graft PCI vs. native vessel PCI. Postprocedural elevation of cardiac markers and elevated ChP IgA were related to a higher cardiac event rate. Left main coronary artery PCI, svg\-PCI and anti\-ChP IgA >12 EIU have been identified as independent predictors of long\-term MACE."]

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