Search for: [Abstract = "rglycemia, as well as episodes of severe hypoglycemia and episodes of ketoacidosis. Similarly to neuropathy, the risk of cognitive impairment is increased by higher HbA1c, higher BMI and smoking. The main mechanism of cognitive impairment is macroangiopathy, which leads to atherosclerosis of cerebral vessels as well as strokes and other morphological changes, especially in the white matter. The effect of angiopathy has been documented, among others, by showing the correlation of cognitive impairment with increased thickness of the intima\-media complex of the common carotid arteries. The frequency and severity of neurological complications in DM1 can be reduced with modern therapeutic options. One of such options is increase in frequency of manual glycaemia measurements and insulin injections. Such treatment results in tighter glycemic control, which, according to meta\-analysis from the Cochrane Library significantly reduces the occurrence of complications. In the last decade therapeutic techniques in DM1 have been further developed with the aim to provide the most physiological level of glycemia. These techniques include continuous glucose monitors \(CGMs\), which are small devices permanently attached to the patient’s body. Using transdermal or subcutaneous sensor CGMs measure blood glucose every one to five minutes and reacts if the glucose level exceeds the normal range, or i"]
